Current Environment: Production

Katherine Michaelis | Medical Services

Programs & Services

Languages

  • English

Katherine Michaelis | Education

Undergraduate School

University of Colorado

Boulder, CO

Graduate School

Oregon Health and Science University

Portland, OR

Medical School

Oregon Health and Science University

Portland, OR

Internship

Pediatrics

Boston Combined Residency Program (BCRP)

Boston, MA

Residency

Pediatrics

Boston Combined Residency Program (BCRP)

Boston, MA

Katherine Michaelis | Publications

  1. Human OX40L-CAR-Tregs target activated antigen-presenting cells and control T cell alloreactivity. Sci Transl Med. 2024 Oct 16; 16(769):eadj9331. View Human OX40L-CAR-Tregs target activated antigen-presenting cells and control T cell alloreactivity. Abstract

  2. COVID-19-Associated Croup in Children. Pediatrics. 2022 06 01; 149(6). View COVID-19-Associated Croup in Children. Abstract

  3. TLR7 is expressed by support cells, but not sensory neurons, in ganglia. J Neuroinflammation. 2021 Sep 16; 18(1):209. View TLR7 is expressed by support cells, but not sensory neurons, in ganglia. Abstract

  4. Chronic cerebral lipocalin 2 exposure elicits hippocampal neuronal dysfunction and cognitive impairment. Brain Behav Immun. 2021 10; 97:102-118. View Chronic cerebral lipocalin 2 exposure elicits hippocampal neuronal dysfunction and cognitive impairment. Abstract

  5. Lipocalin 2 mediates appetite suppression during pancreatic cancer cachexia. Nat Commun. 2021 04 06; 12(1):2057. View Lipocalin 2 mediates appetite suppression during pancreatic cancer cachexia. Abstract

  6. Hospitalization Rates and Characteristics of Children Aged <18 Years Hospitalized with Laboratory-Confirmed COVID-19 - COVID-NET, 14 States, March 1-July 25, 2020. MMWR Morb Mortal Wkly Rep. 2020 Aug 14; 69(32):1081-1088. View Hospitalization Rates and Characteristics of Children Aged <18 Years Hospitalized with Laboratory-Confirmed COVID-19 - COVID-NET, 14 States, March 1-July 25, 2020. Abstract

  7. Circulating myeloid cells invade the central nervous system to mediate cachexia during pancreatic cancer. Elife. 2020 05 11; 9. View Circulating myeloid cells invade the central nervous system to mediate cachexia during pancreatic cancer. Abstract

  8. Microglia in the hypothalamus respond to tumor-derived factors and are protective against cachexia during pancreatic cancer. Glia. 2020 07; 68(7):1479-1494. View Microglia in the hypothalamus respond to tumor-derived factors and are protective against cachexia during pancreatic cancer. Abstract

  9. Publisher Correction: The TLR7/8 agonist R848 remodels tumor and host responses to promote survival in pancreatic cancer. Nat Commun. 2019 Nov 15; 10(1):5257. View Publisher Correction: The TLR7/8 agonist R848 remodels tumor and host responses to promote survival in pancreatic cancer. Abstract

  10. The TLR7/8 agonist R848 remodels tumor and host responses to promote survival in pancreatic cancer. Nat Commun. 2019 10 15; 10(1):4682. View The TLR7/8 agonist R848 remodels tumor and host responses to promote survival in pancreatic cancer. Abstract

  11. Persistent Toll-like receptor 7 stimulation induces behavioral and molecular innate immune tolerance. Brain Behav Immun. 2019 11; 82:338-353. View Persistent Toll-like receptor 7 stimulation induces behavioral and molecular innate immune tolerance. Abstract

  12. MyD88 signalling is critical in the development of pancreatic cancer cachexia. J Cachexia Sarcopenia Muscle. 2019 04; 10(2):378-390. View MyD88 signalling is critical in the development of pancreatic cancer cachexia. Abstract

  13. Increasing lean muscle mass in mice via nanoparticle-mediated hepatic delivery of follistatin mRNA. Theranostics. 2018; 8(19):5276-5288. View Increasing lean muscle mass in mice via nanoparticle-mediated hepatic delivery of follistatin mRNA. Abstract

  14. TRIF is a key inflammatory mediator of acute sickness behavior and cancer cachexia. Brain Behav Immun. 2018 10; 73:364-374. View TRIF is a key inflammatory mediator of acute sickness behavior and cancer cachexia. Abstract

  15. Establishment and characterization of a novel murine model of pancreatic cancer cachexia. J Cachexia Sarcopenia Muscle. 2017 Oct; 8(5):824-838. View Establishment and characterization of a novel murine model of pancreatic cancer cachexia. Abstract

  16. A distinct brain pathway links viral RNA exposure to sickness behavior. Sci Rep. 2016 07 20; 6:29885. View A distinct brain pathway links viral RNA exposure to sickness behavior. Abstract

  17. The central role of hypothalamic inflammation in the acute illness response and cachexia. Semin Cell Dev Biol. 2016 06; 54:42-52. View The central role of hypothalamic inflammation in the acute illness response and cachexia. Abstract

  18. Sustained hyperoxia-induced NF-?B activation improves survival and preserves lung development in neonatal mice. Am J Physiol Lung Cell Mol Physiol. 2014 Jun 15; 306(12):L1078-89. View Sustained hyperoxia-induced NF-?B activation improves survival and preserves lung development in neonatal mice. Abstract

  19. I?Bß-mediated NF-?B activation confers protection against hyperoxic lung injury. Am J Respir Cell Mol Biol. 2014 Feb; 50(2):429-38. View I?Bß-mediated NF-?B activation confers protection against hyperoxic lung injury. Abstract

  20. The NF-?B inhibitory proteins I?Ba and I?Bß mediate disparate responses to inflammation in fetal pulmonary endothelial cells. J Immunol. 2013 Mar 15; 190(6):2913-23. View The NF-?B inhibitory proteins I?Ba and I?Bß mediate disparate responses to inflammation in fetal pulmonary endothelial cells. Abstract

  21. Reprimo (RPRM) is a novel tumor suppressor in pituitary tumors and regulates survival, proliferation, and tumorigenicity. Endocrinology. 2012 Jul; 153(7):2963-73. View Reprimo (RPRM) is a novel tumor suppressor in pituitary tumors and regulates survival, proliferation, and tumorigenicity. Abstract

  22. Nuclear factor-?B (NF-?B) inhibitory protein I?Bß determines apoptotic cell death following exposure to oxidative stress. J Biol Chem. 2012 Feb 24; 287(9):6230-9. View Nuclear factor-?B (NF-?B) inhibitory protein I?Bß determines apoptotic cell death following exposure to oxidative stress. Abstract

  23. Identification of growth arrest and DNA-damage-inducible gene beta (GADD45beta) as a novel tumor suppressor in pituitary gonadotrope tumors. Endocrinology. 2011 Oct; 152(10):3603-13. View Identification of growth arrest and DNA-damage-inducible gene beta (GADD45beta) as a novel tumor suppressor in pituitary gonadotrope tumors. Abstract

  24. Giant pituitary adenomas: pathologic-radiographic correlations and lack of role for p53 and MIB-1 labeling. Am J Surg Pathol. 2011 Aug; 35(8):1204-13. View Giant pituitary adenomas: pathologic-radiographic correlations and lack of role for p53 and MIB-1 labeling. Abstract

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