Sarah Low, MD
Associate in Pain Medicine, Department of Anesthesia, Critical Care and Pain Medicine
Instructor, Harvard Medical School
Image
NPI
1265895841
Locations
Sarah Low, MD
Associate in Pain Medicine, Department of Anesthesia, Critical Care and Pain Medicine
Instructor, Harvard Medical School
Medical Services
Languages
English
Education
Undergraduate School
University of Arizona
Tucson
AZ
Medical School
University of Arizona College of Medicine
Tucson
AZ
Internship
Internal Medicine
University of Arizona Banner Health
Tucson
AZ
Residency
Anesthesiology
Massachusetts General Hospital
Boston
MA
Fellowship
Pediatric Anesthesiology
Massachusetts General Hospital
Boston
MA
Fellowship
Pediatric Pain Medicine
Boston Children’s Hospital
Boston
MA
Certifications
American Board of Anesthesiology (Pediatric Anesthesiology)
American Board of Anesthesiology (General)
American Board of Anesthesiology (Pain Medicine)
Publications
Awake Combined Spinal Caudal Anesthesia for Longer-Duration Urologic Surgery in Infants: A Case Series. View Abstract
Evaluating the impact of broadband access and internet use in a small underserved rural community. View Abstract
Broadband adoption and availability: Impacts on rural employment during COVID-19. View Abstract
Multisystem effects of COVID-19: a concise review for practitioners. View Abstract
Ultrasound Teleguidance to Reduce Healthcare Worker Exposure to Coronavirus Disease 2019. View Abstract
Functional Divergence of Delta and Mu Opioid Receptor Organization in CNS Pain Circuits. View Abstract
Complex management of a patient with refractory primary erythromelalgia lacking a SCN9A mutation. View Abstract
A Brainstem-Spinal Cord Inhibitory Circuit for Mechanical Pain Modulation by GABA and Enkephalins. View Abstract
Loss of µ opioid receptor signaling in nociceptors, but not microglia, abrogates morphine tolerance without disrupting analgesia. View Abstract
GINIP, a Gai-interacting protein, functions as a key modulator of peripheral GABAB receptor-mediated analgesia. View Abstract
The Netrin-1 receptor DCC is a regulator of maladaptive responses to chronic morphine administration. View Abstract
VGLUT2 expression in primary afferent neurons is essential for normal acute pain and injury-induced heat hypersensitivity. View Abstract
Locations