Katherine Michaelis, MD, PhD

Clinical Fellow, Division of Immunology
Katherine Michaelis, MD, PhD
Phone 617-355-6117
Fax 617-730-4813
NPI 1699351270
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Katherine Michaelis, MD, PhD

Clinical Fellow, Division of Immunology

Medical Services

Programs & Services
Allergy and Asthma Program
Immunology Program
Immunology
Languages
English
Education
Undergraduate School
University of Colorado
Boulder
CO
Graduate School
Oregon Health and Science University
Portland
OR
Medical School
Oregon Health and Science University
Portland
OR
Internship
Pediatrics
Boston Combined Residency Program (BCRP)
Boston
MA
Residency
Pediatrics
Boston Combined Residency Program (BCRP)
Boston
MA

Publications

Human OX40L-CAR-Tregs target activated antigen-presenting cells and control T cell alloreactivity. View Abstract
COVID-19-Associated Croup in Children. View Abstract
TLR7 is expressed by support cells, but not sensory neurons, in ganglia. View Abstract
Chronic cerebral lipocalin 2 exposure elicits hippocampal neuronal dysfunction and cognitive impairment. View Abstract
Lipocalin 2 mediates appetite suppression during pancreatic cancer cachexia. View Abstract
Hospitalization Rates and Characteristics of Children Aged <18 Years Hospitalized with Laboratory-Confirmed COVID-19 - COVID-NET, 14 States, March 1-July 25, 2020. View Abstract
Circulating myeloid cells invade the central nervous system to mediate cachexia during pancreatic cancer. View Abstract
Microglia in the hypothalamus respond to tumor-derived factors and are protective against cachexia during pancreatic cancer. View Abstract
Publisher Correction: The TLR7/8 agonist R848 remodels tumor and host responses to promote survival in pancreatic cancer. View Abstract
The TLR7/8 agonist R848 remodels tumor and host responses to promote survival in pancreatic cancer. View Abstract
Persistent Toll-like receptor 7 stimulation induces behavioral and molecular innate immune tolerance. View Abstract
MyD88 signalling is critical in the development of pancreatic cancer cachexia. View Abstract
Increasing lean muscle mass in mice via nanoparticle-mediated hepatic delivery of follistatin mRNA. View Abstract
TRIF is a key inflammatory mediator of acute sickness behavior and cancer cachexia. View Abstract
Establishment and characterization of a novel murine model of pancreatic cancer cachexia. View Abstract
A distinct brain pathway links viral RNA exposure to sickness behavior. View Abstract
The central role of hypothalamic inflammation in the acute illness response and cachexia. View Abstract
Sustained hyperoxia-induced NF-?B activation improves survival and preserves lung development in neonatal mice. View Abstract
I?Bß-mediated NF-?B activation confers protection against hyperoxic lung injury. View Abstract
The NF-?B inhibitory proteins I?Ba and I?Bß mediate disparate responses to inflammation in fetal pulmonary endothelial cells. View Abstract
Reprimo (RPRM) is a novel tumor suppressor in pituitary tumors and regulates survival, proliferation, and tumorigenicity. View Abstract
Nuclear factor-?B (NF-?B) inhibitory protein I?Bß determines apoptotic cell death following exposure to oxidative stress. View Abstract
Identification of growth arrest and DNA-damage-inducible gene beta (GADD45beta) as a novel tumor suppressor in pituitary gonadotrope tumors. View Abstract
Giant pituitary adenomas: pathologic-radiographic correlations and lack of role for p53 and MIB-1 labeling. View Abstract
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